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Cartalax

Aliases: Ala-Glu-Asp · AED (cartilage)

Last verified: 2026-09-24

Research chemicalLimited evidenceBioregulatorsLow interest

The short version

Cartalax belongs to the 'bioregulator' family — tripeptides for which one institution claims to be tissue signals. The classic pattern: a few older publications with statistically modest samples, no independent replication, no RCTs. Unlike Epitalon, there is at least biological plausibility here (three-amino-acid peptides can have cell effects in culture), but the path from chondrocyte culture to 'treating human joints' is longer than any available study travels. Risk is low; evidence is even lower.

Identity & type

Molecular type
bioregulator peptide
Sequence / structure
Ala-Glu-Asp (3 aa)
Molecular weight
~375 Da
Origin
Synthetic tripeptide from the Soviet/Russian bioregulator program (St. Petersburg).

Mechanism of action

A tripeptide (Ala-Glu-Asp) from the Khavinson bioregulator program; claimed to act as a 'signaling' peptide that modulates chondrocyte differentiation and collagen II synthesis — publications come mostly from the same author group.

not confirmed in humans

Dosing & routes

Official / clinical context

No approved official document exists.

Regulator-approved labeling and published study designs — never a recommendation.

Community-reported practice

Rarely used outside the former Soviet sphere; appears in communities among older users with joint complaints, with stories of 'mild improvement' — without objective measures.

Unverified self-reports. Not medical advice. Not endorsement.

Protocol — official vs community

Official / label

No approved protocol exists. Cartalax is a research peptide bioregulator (Ala-Glu-Asp) with no registered drug product in any major market.

Duration:

Chondrocyte-differentiation and collagen-II claims come mostly from the originating author group; no standardized trial data.

Community (anecdotal)

5–10 mg SC daily for 10–15 days, or 1–2 mg orally per the program pattern.

Cycle:
1–2 courses per year, repeated.
Break:
Months between courses.

Follows the program-pattern variant of the shared course structure; only the cartilage/joint targeting claim distinguishes it from the rest of the series.

Human evidence

No controlled human trials; only small reports from the same source claiming an effect.

Preclinical evidence

Limited and independently unverified: cell cultures and aged-animal models from one group.

Known risks

  • No significant adverse effects documentedcharacterized
  • Opportunity cost: delaying real joint treatmenttheoretical

Teal: characterized in clinical/labeling contexts. Amber: theoretical or reported outside controlled settings.

Unknowns & evidence gaps

  • Everything — no independent human data

Frequently asked questions

References

  1. Khavinson group publications on cartilage peptide bioregulators (single-source literature)